Given its role in cancer development, CagA represents a potential target for therapeutic intervention. Strategies could include the development of small molecules or antibodies that inhibit CagA's interactions with host cell proteins or its phosphorylation. Furthermore, vaccination against H. pylori, particularly targeting CagA, could reduce the incidence of infection and consequently, the risk of gastric cancer. Some research is also focused on the use of probiotics and antibiotics to eradicate H. pylori infection and reduce its pathogenic effects.
